@Hugin2017 read all of Chudov's article and wanted to present three hypotheses as to why the death rate is spiking. I do not believe we are moving towards a "super Coronachan" variant as this would go in the face of hundreds of years of normal coronaviridae evolution, which always seem to settle on a 'less-lethal' variant

1. Constant infection and re-infection with ever-evolving variants without development of lasting immunity (due to OAS) resulting in eventual immunologic cell destruction and massive systemic spread of Chinkflu viral load, causing multi-system organ failure (call this the ADE hypothesis). Most likely to be found in fagcinated and repeatedly boosted individuals

Likelihood: Medium-high

2. The same infection-reinfection cycle causing critical chronic depletion of cytokines and an immunoglobulin shift towards "tolerating" disease rather than "eliminating" it. This leaves the affected individual vulnerable to other opportunistic infections. Call this the VAIDS or fagcine-GRIDS hypothesis.

Likelihood: Medium

3. Chronic infection (OAS) with coronachan variants causing repeated spike-protein related damage to organ systems, particularly the cardiopulmonary system. In addition to what initial fagcination caused. Call this the 'fagcine-plus' hypothesis.

Likelihood: Low

Let me know if this is helpful