@Hugin2017 asked me to write a brief follow-up explaining the concept of "Original Antigenic Sin" (OAS) and why the "vaccines" (gene therapy) are not only completely ineffective at this point, but frankly damaging and counterproductive. So here we go again:
OAS isn't a recent concept, we've known about it since 1950 ("Hoskins effect"). It has to do with the antibodies produced by B-cells from your immune system upon first encountering a viral epitope (portion of the virus that triggers an immune response). Now, a small percentage of these B-cells will become 'memory B-cells' after you clear the infection, which is to say they will not undergo normal cell turnover through natural programmed death mechanisms; they will reside in your body, potentially for decades, and will undergo reactivation immediately upon you re-encountering the infectious agent that they were developed against. The body does this so that you can clear the infection faster the second time around with less resource consumption.
So far so good. However . . .
These memory B-cells deliberately INHIBIT your naïve B-cells from producing a fresh de novo antibody response against other viral epitopes. This is the 'original sin' referred to in the nickname. And this is a problem if the viral epitope you had previously been exposed to (like, say, an mRNA gene therapy spike protein) has undergone 'antigenic drift' via mutations, because you are now preferentially producing suboptimal antibodies against the infection, and you will take LONGER to clear it than you would have if you had just freshly been exposed. 'Boosting' your immune system with the same ineffective epitope does nothing but worsen this effect, as you are just inhibiting your naïve B-cells from taking over and fixing the problem.
Good thing WuFlu isn't part of a viral family notorious for rapid mutation and antigenic drift, especially under artificial selection press-
Oh, yeah
OAS isn't a recent concept, we've known about it since 1950 ("Hoskins effect"). It has to do with the antibodies produced by B-cells from your immune system upon first encountering a viral epitope (portion of the virus that triggers an immune response). Now, a small percentage of these B-cells will become 'memory B-cells' after you clear the infection, which is to say they will not undergo normal cell turnover through natural programmed death mechanisms; they will reside in your body, potentially for decades, and will undergo reactivation immediately upon you re-encountering the infectious agent that they were developed against. The body does this so that you can clear the infection faster the second time around with less resource consumption.
So far so good. However . . .
These memory B-cells deliberately INHIBIT your naïve B-cells from producing a fresh de novo antibody response against other viral epitopes. This is the 'original sin' referred to in the nickname. And this is a problem if the viral epitope you had previously been exposed to (like, say, an mRNA gene therapy spike protein) has undergone 'antigenic drift' via mutations, because you are now preferentially producing suboptimal antibodies against the infection, and you will take LONGER to clear it than you would have if you had just freshly been exposed. 'Boosting' your immune system with the same ineffective epitope does nothing but worsen this effect, as you are just inhibiting your naïve B-cells from taking over and fixing the problem.
Good thing WuFlu isn't part of a viral family notorious for rapid mutation and antigenic drift, especially under artificial selection press-
Oh, yeah
K, here goes:
The way your body destroys viral infections depends on an initial antibody response (normally produced via B-cells) "tagging" the virus as foreign so that killer white blood cells (T-cells/macrophages) from your immune system will recognize the antibody tags, kill virus-infected cells, absorb the viral particles, and destroy them.
However, if the initial antibody "tags" are improperly formed it is possible that the immune cells will absorb the viral agent but be unable to destroy it (it's a complex sequential process that requires numerous receptors to access the antibody "tags").
So, for example, if you have received an experimental gene therapy that produces inferior antibodies rather than the normal ones unvaccinated people's B-cells produce, this is what can happen if you are exposed to even a slight variant of the initial virus you were "vaccinated" against.
This is very, very bad because it means that the initial viral load you were infected with, however small, is now shielded within your own white blood cells. The virus will replicate freely. Your infected immune cells will circulate throughout your body. Eventually, the intracellular viral load will overwhelm the immune cells and cause them to rupture. And then you very rapidly go from relatively normal to critically ill and septic because this is happening on a massive scale, with a much larger viral load than you were initially exposed to suddenly overwhelming your defenses as the immune cells die, resulting in multiple organ systems shutting down at once. Survival rates from multi-system organ failure are dismal.
This is why mRNA "vaccines" (gene therapy) were never approved prior to 2020. The animal studies kept showing this lethal effect.
It's called "antibody-dependent enhancement". We're going to be hearing about it a lot.
The way your body destroys viral infections depends on an initial antibody response (normally produced via B-cells) "tagging" the virus as foreign so that killer white blood cells (T-cells/macrophages) from your immune system will recognize the antibody tags, kill virus-infected cells, absorb the viral particles, and destroy them.
However, if the initial antibody "tags" are improperly formed it is possible that the immune cells will absorb the viral agent but be unable to destroy it (it's a complex sequential process that requires numerous receptors to access the antibody "tags").
So, for example, if you have received an experimental gene therapy that produces inferior antibodies rather than the normal ones unvaccinated people's B-cells produce, this is what can happen if you are exposed to even a slight variant of the initial virus you were "vaccinated" against.
This is very, very bad because it means that the initial viral load you were infected with, however small, is now shielded within your own white blood cells. The virus will replicate freely. Your infected immune cells will circulate throughout your body. Eventually, the intracellular viral load will overwhelm the immune cells and cause them to rupture. And then you very rapidly go from relatively normal to critically ill and septic because this is happening on a massive scale, with a much larger viral load than you were initially exposed to suddenly overwhelming your defenses as the immune cells die, resulting in multiple organ systems shutting down at once. Survival rates from multi-system organ failure are dismal.
This is why mRNA "vaccines" (gene therapy) were never approved prior to 2020. The animal studies kept showing this lethal effect.
It's called "antibody-dependent enhancement". We're going to be hearing about it a lot.
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