DOES THE VACCINE TARGET CHILDREN?
IS THIS WHY THEY ARE DRIVING IT SO HARD FOR KIDS?
GET ADULTS NORMALIZED FIRST?
THEN TO THE CHILDREN?
YOU SHOULD READ THIS.
CVID v. COVID
THERE'S EVEN A CVID2
Question: Did they program CVID into an mRNA vaccine so that it would cause immunodeficiency through forced gene mutations and allow them to blame it on the virus used to justify the same vaccine? Just missing an O.
First, study the CVID image closely studying the nature of the disease and noting the COVID overlaps.
Then read the second for the same noting the causes.
From the second image, latch on to the TNFRSF13B gene,
"In about 10 percent of cases, a genetic cause for CVID is known. Mutations in at least 13 genes have been associated with CVID. The most frequent mutations occur in the TNFRSF13B gene."
Now read the third image and from it, consider this relative to the TNFRSF13B gene. We'll start with the title,
"TBK1 and TNFRSF13B mutations and an autoinflammatory disease in a child with lethal COVID-19"
From the same,
"Among children, severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infections are typically mild. Here, we describe the case of a 3.5-year-old girl with an unusually severe presentation of coronavirus disease (COVID-19). The child had an autoinflammatory disorder of unknown etiology, which had been treated using prednisolone and methotrexate, and her parents were half cousins of Turkish descent. After 5 days of nonspecific viral infection symptoms, tonic-clonic seizures occurred followed by acute cardiac insufficiency, multi-organ insufficiency, and ultimate death. Trio exome sequencing identified a homozygous splice-variant in the gene TBK1, and a homozygous missense variant in the gene TNFRSF13B."
Further, "Of these, one homozygous variant in the genes TBK1 and TNFRSF13B, respectively (see Table Table2)2) were considered likely causal factors for the severe COVID-19 disease course observed in the present case."
From the same, "Additionally, reduced penetrance and variable expressivity have been observed in homozygous carriers of the reported TNFRSF13B variant for CVID2."
"The largest study of comorbid CVID/COVID-19 to date included ten patients. Due to the relatively mild disease course observed in all cases, the authors concluded that this population showed no increased risk for a severe COVID-19 disease course26. In summary, the TNFRSF13B-variant may have contributed to the fulminant SARS-CoV-2 infection and to the nonspecific, preexisting autoinflammatory disease observed in the present case."
So, I'll ask again because I don't know. I've never heard of CVID or CVID2 and I've spent thousands of hours running this stuff down.
DOES THE VACCINE TARGET CHILDREN?
Better find out and now.
#RICO
https://www.niaid.nih.gov/diseases-conditions/common-variable-immunodeficiency-cvid
https://medlineplus.gov/genetics/condition/common-variable-immune-deficiency/
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8249618/
IS THIS WHY THEY ARE DRIVING IT SO HARD FOR KIDS?
GET ADULTS NORMALIZED FIRST?
THEN TO THE CHILDREN?
YOU SHOULD READ THIS.
CVID v. COVID
THERE'S EVEN A CVID2
Question: Did they program CVID into an mRNA vaccine so that it would cause immunodeficiency through forced gene mutations and allow them to blame it on the virus used to justify the same vaccine? Just missing an O.
First, study the CVID image closely studying the nature of the disease and noting the COVID overlaps.
Then read the second for the same noting the causes.
From the second image, latch on to the TNFRSF13B gene,
"In about 10 percent of cases, a genetic cause for CVID is known. Mutations in at least 13 genes have been associated with CVID. The most frequent mutations occur in the TNFRSF13B gene."
Now read the third image and from it, consider this relative to the TNFRSF13B gene. We'll start with the title,
"TBK1 and TNFRSF13B mutations and an autoinflammatory disease in a child with lethal COVID-19"
From the same,
"Among children, severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infections are typically mild. Here, we describe the case of a 3.5-year-old girl with an unusually severe presentation of coronavirus disease (COVID-19). The child had an autoinflammatory disorder of unknown etiology, which had been treated using prednisolone and methotrexate, and her parents were half cousins of Turkish descent. After 5 days of nonspecific viral infection symptoms, tonic-clonic seizures occurred followed by acute cardiac insufficiency, multi-organ insufficiency, and ultimate death. Trio exome sequencing identified a homozygous splice-variant in the gene TBK1, and a homozygous missense variant in the gene TNFRSF13B."
Further, "Of these, one homozygous variant in the genes TBK1 and TNFRSF13B, respectively (see Table Table2)2) were considered likely causal factors for the severe COVID-19 disease course observed in the present case."
From the same, "Additionally, reduced penetrance and variable expressivity have been observed in homozygous carriers of the reported TNFRSF13B variant for CVID2."
"The largest study of comorbid CVID/COVID-19 to date included ten patients. Due to the relatively mild disease course observed in all cases, the authors concluded that this population showed no increased risk for a severe COVID-19 disease course26. In summary, the TNFRSF13B-variant may have contributed to the fulminant SARS-CoV-2 infection and to the nonspecific, preexisting autoinflammatory disease observed in the present case."
So, I'll ask again because I don't know. I've never heard of CVID or CVID2 and I've spent thousands of hours running this stuff down.
DOES THE VACCINE TARGET CHILDREN?
Better find out and now.
#RICO
https://www.niaid.nih.gov/diseases-conditions/common-variable-immunodeficiency-cvid
https://medlineplus.gov/genetics/condition/common-variable-immune-deficiency/
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8249618/
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